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Pregled bibliografske jedinice broj: 667208

Endocytic traffick jam in Niemann Pick type C disease – a cause of increased Alzheimer’s amyloid-β peptide formation


Katušić Hećimović, Silva
Endocytic traffick jam in Niemann Pick type C disease – a cause of increased Alzheimer’s amyloid-β peptide formation // Gordon Research Conference – Lysosomal Diseases
Galveston (TX), Sjedinjene Američke Države, 2011. (pozvano predavanje, međunarodna recenzija, neobjavljeni rad, znanstveni)


CROSBI ID: 667208 Za ispravke kontaktirajte CROSBI podršku putem web obrasca

Naslov
Endocytic traffick jam in Niemann Pick type C disease – a cause of increased Alzheimer’s amyloid-β peptide formation

Autori
Katušić Hećimović, Silva

Vrsta, podvrsta i kategorija rada
Sažeci sa skupova, neobjavljeni rad, znanstveni

Skup
Gordon Research Conference – Lysosomal Diseases

Mjesto i datum
Galveston (TX), Sjedinjene Američke Države, 23.01.2011. - 28.01.2011

Vrsta sudjelovanja
Pozvano predavanje

Vrsta recenzije
Međunarodna recenzija

Ključne riječi
Alzheimer's disease; APP; amyloid-beta; lysosomal storage diseases; NPC1

Sažetak
We showed that cholesterol accumulation due to NPC1-loss of function causes accumulation of both APP and BACE1, the two key proteins in the pathogenesis of Alzheimer's disease, within endocytic pathway.

Izvorni jezik
Engleski

Znanstvena područja
Biologija, Temeljne medicinske znanosti



POVEZANOST RADA


Projekti:
098-0982522-2525 - Mehanizam djelovanja kolesterola u nastanku Alzheimerove bolesti (Katušić Hećimović, Silva, MZOS ) ( CroRIS)

Ustanove:
Institut "Ruđer Bošković", Zagreb


Citiraj ovu publikaciju:

Katušić Hećimović, Silva
Endocytic traffick jam in Niemann Pick type C disease – a cause of increased Alzheimer’s amyloid-β peptide formation // Gordon Research Conference – Lysosomal Diseases
Galveston (TX), Sjedinjene Američke Države, 2011. (pozvano predavanje, međunarodna recenzija, neobjavljeni rad, znanstveni)
Katušić Hećimović, S. (2011) Endocytic traffick jam in Niemann Pick type C disease – a cause of increased Alzheimer’s amyloid-β peptide formation. U: Gordon Research Conference – Lysosomal Diseases.
@article{article, author = {Katu\v{s}i\'{c} He\'{c}imovi\'{c}, Silva}, year = {2011}, keywords = {Alzheimer's disease, APP, amyloid-beta, lysosomal storage diseases, NPC1}, title = {Endocytic traffick jam in Niemann Pick type C disease – a cause of increased Alzheimer’s amyloid-β peptide formation}, keyword = {Alzheimer's disease, APP, amyloid-beta, lysosomal storage diseases, NPC1}, publisherplace = {Galveston (TX), Sjedinjene Ameri\v{c}ke Dr\v{z}ave} }
@article{article, author = {Katu\v{s}i\'{c} He\'{c}imovi\'{c}, Silva}, year = {2011}, keywords = {Alzheimer's disease, APP, amyloid-beta, lysosomal storage diseases, NPC1}, title = {Endocytic traffick jam in Niemann Pick type C disease – a cause of increased Alzheimer’s amyloid-β peptide formation}, keyword = {Alzheimer's disease, APP, amyloid-beta, lysosomal storage diseases, NPC1}, publisherplace = {Galveston (TX), Sjedinjene Ameri\v{c}ke Dr\v{z}ave} }




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