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Pregled bibliografske jedinice broj: 1263322

EReply to: Atrial apoptosis and fibrosis adversely affect atrial conduit, reservoir and contractile functions

Gasparovic, Hrvoje; Cikes, Maja; Kopjar, Tomislav; Biocina, Bojan
eReply to: Atrial apoptosis and fibrosis adversely affect atrial conduit, reservoir and contractile functions // Interactive CardioVascular and Thoracic Surgery, 19 (2014), 2; 2302-231 doi:10.1093/icvts/ivu192 (međunarodna recenzija, pismo uredniku, znanstveni)

CROSBI ID: 1263322 Za ispravke kontaktirajte CROSBI podršku putem web obrasca

Naslov
EReply to: Atrial apoptosis and fibrosis adversely affect atrial conduit, reservoir and contractile functions

Autori
Gasparovic, Hrvoje ; Cikes, Maja ; Kopjar, Tomislav ; Biocina, Bojan

Izvornik
Interactive CardioVascular and Thoracic Surgery (1569-9293) 19 (2014), 2; 2302-231

Vrsta, podvrsta i kategorija rada
Radovi u časopisima, pismo uredniku, znanstveni

Ključne riječi
apoptosis conduit implant atrium fibrosis muscle contraction

Sažetak
We appreciate the insightful comments made by Bockeria et al. on our recent article. We completely agree with the authors that placing histopathological changes into clinical context is important. The paucity of data correlating the magnitude of adverse cellular remodelling with clinical outcomes among patients undergoing atrial fibrillation (AF) surgery mirrors the logistical constraints of such endeavours. In our study, we investigated the impact of histological changes on echocardiographic descriptors of atrial conduit, reservoir and contractile functions. We found that both atrial apoptosis and fibrosis yielded significant declines across all facets of atrial function.

Izvorni jezik
Engleski

Znanstvena područja
Biotehnologija u biomedicini (prirodno područje, biomedicina i zdravstvo, biotehničko područje)



POVEZANOST RADA

Ustanove:
Medicinski fakultet, Zagreb,
Klinički bolnički centar Zagreb

Profili:

Avatar Url Bojan Biočina (autor)

Avatar Url Tomislav Kopjar (autor)

Avatar Url Maja Čikeš (autor)

Avatar Url Hrvoje Gašparović (autor)

Poveznice na cjeloviti tekst rada:

doi

Citiraj ovu publikaciju:

Gasparovic, Hrvoje; Cikes, Maja; Kopjar, Tomislav; Biocina, Bojan
eReply to: Atrial apoptosis and fibrosis adversely affect atrial conduit, reservoir and contractile functions // Interactive CardioVascular and Thoracic Surgery, 19 (2014), 2; 2302-231 doi:10.1093/icvts/ivu192 (međunarodna recenzija, pismo uredniku, znanstveni)
Gasparovic, H., Cikes, M., Kopjar, T. & Biocina, B. (2014) eReply to: Atrial apoptosis and fibrosis adversely affect atrial conduit, reservoir and contractile functions. Interactive CardioVascular and Thoracic Surgery, 19 (2), 2302-231 doi:10.1093/icvts/ivu192.
@article{article, author = {Gasparovic, Hrvoje and Cikes, Maja and Kopjar, Tomislav and Biocina, Bojan}, year = {2014}, pages = {2302-231}, DOI = {10.1093/icvts/ivu192}, keywords = {apoptosis conduit implant atrium fibrosis muscle contraction}, journal = {Interactive CardioVascular and Thoracic Surgery}, doi = {10.1093/icvts/ivu192}, volume = {19}, number = {2}, issn = {1569-9293}, title = {eReply to: Atrial apoptosis and fibrosis adversely affect atrial conduit, reservoir and contractile functions}, keyword = {apoptosis conduit implant atrium fibrosis muscle contraction} }
@article{article, author = {Gasparovic, Hrvoje and Cikes, Maja and Kopjar, Tomislav and Biocina, Bojan}, year = {2014}, pages = {2302-231}, DOI = {10.1093/icvts/ivu192}, keywords = {apoptosis conduit implant atrium fibrosis muscle contraction}, journal = {Interactive CardioVascular and Thoracic Surgery}, doi = {10.1093/icvts/ivu192}, volume = {19}, number = {2}, issn = {1569-9293}, title = {eReply to: Atrial apoptosis and fibrosis adversely affect atrial conduit, reservoir and contractile functions}, keyword = {apoptosis conduit implant atrium fibrosis muscle contraction} }

Časopis indeksira:


  • Current Contents Connect (CCC)
  • Web of Science Core Collection (WoSCC)
    • Science Citation Index Expanded (SCI-EXP)
    • SCI-EXP, SSCI i/ili A&HCI
  • Scopus
  • MEDLINE

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