A Homozygous Dab1−/− Is a Potential Novel Cause of Autosomal Recessive Congenital Anomalies of the Mice Kidney and Urinary Tract (CROSBI ID 293811)
Prilog u časopisu | izvorni znanstveni rad | međunarodna recenzija
Podaci o odgovornosti
Racetin, Anita ; Filipović, Natalija ; Lozić, Mirela ; Ogata, Masaki ; Larissa, Gudelj Ensor ; Kelam, Nela ; Kovačević, Petra ; Watanabe, Koichiro ; Katsuyama, Yu ; Saraga-Babić, Mirna ; Glavina Durdov, Merica ; Vukojević, Katarina
engleski
A Homozygous Dab1−/− Is a Potential Novel Cause of Autosomal Recessive Congenital Anomalies of the Mice Kidney and Urinary Tract
This study aimed to explore morphology changes in the kidneys of Dab1−/− (yotari) mice, as well as expression patterns of reelin, NOTCH2, LC3B, and cleaved caspase3 (CASP3) proteins, as potential determinants of normal kidney formation and function. We assumed that Dab1 functional inactivation may cause disorder in a wide spectrum of congenital anomalies of the kidney and urinary tract (CAKUT).
yotari ; kidney function ; postnatal kidney development ; immunofluorescence staining ; transmission electron microscopy
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Podaci o izdanju
Povezanost rada
Temeljne medicinske znanosti